Wilson’s Disease (WD) is a recessively inherited autosomal disease related to mutations in the ATP7B gene caused by the accumulation of
excess copper in the body, particularly in the liver, brain, and kidney.
It especially affects children and young adults, but it can occur before
three and after 40 years of age1. The clinical profile of hepatic manifestations may vary from asymptomatic biochemical abnormalities
and steatosis to acute hepatitis, acute liver failure, chronic hepatitis,